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Wehmeyer, Michael L.. Ed.; Brown, Ivan, Ed.; Percy, Maire, Ed.; Fung, W. L. Alan, Ed.; Shogren, Karrie A., Ed. – Brookes Publishing Company, 2017
The trusted core disability textbook gets a comprehensive update in this second edition, now thoroughly revised to include all the critical topics today's professionals need to know about as they work with people who have intellectual and developmental disabilities. Brought to you by a new team of world-renowned experts and contributors, this…
Descriptors: Intellectual Disability, Developmental Disabilities, Student Development, Etiology
Hirschkorn, Mark; Geelan, David – Alberta Journal of Educational Research, 2008
The issue of the "research-practice gap"--the problematic relationship between research in education and educational practice--has been widely reported in the literature. This critical literature review explores some of the causes and features of the gap and suggests some possible approaches for addressing it. These solutions involve changes in…
Descriptors: Theory Practice Relationship, Educational Practices, Educational Research, Literature Reviews

Sahley, Tony L.; Panksepp, Jaak – Journal of Autism and Developmental Disorders, 1987
The paper summarizes experimental evidence supporting a neurological theory which posits that autism, at least partially, represents a disruptive overactivation of hypersensitization of neurohormone systems in the brain, such as brain opioids. These substances modulate social-emotional processes. (Author/DB)
Descriptors: Autism, Biochemistry, Etiology, Neurological Impairments

Smith, Shelley D.; Kelley, Philip M.; Askew, James W.; Hoover, Denise M.; Deffenbacher, Karen E.; Gayan, Javier; Brower, Amy M.; Olson, Richard K. – Journal of Learning Disabilities, 2001
This study investigated the role of the myelin oligodendrocye glycoportein (MOG) gene to reading disability in 22 children with dyslexia and 96 random controls. Results suggest that, although alleles in the MOG gene may be in linkage disequilibrium with a locus that contributes to reading disability, the MOG gene itself is probably not involved.…
Descriptors: Children, DNA, Dyslexia, Etiology

Pritchard, Walter S.; And Others – Journal of Autism and Developmental Disorders, 1987
The hypothesis that autistics may experience a degree of stimulus overload was supported by an experiment in which visual event-related potentials and cognitive effects were recorded for five male autistic children (ages 6-14 years) and five matched controls. (DB)
Descriptors: Autism, Children, Etiology, Neurology

Minshew, Nancy J. – Journal of Autism and Developmental Disorders, 1996
This paper summarizes results of research on functional and structural abnormalities of the brain in autism. The current concept of causation is seen to involve multiple biologic levels. A consistent profile of brain function and dysfunction across methods has been found and specific neuropathologic findings have been found; but some research…
Descriptors: Anatomy, Autism, Etiology, Neurological Impairments

Filipek, Pauline A. – Journal of Autism and Developmental Disorders, 1996
This review of magnetic resonance imaging (MRI) in autism addresses three questions: (1) what structural anomalies of the brain have been identified by MRI? (2) why are these collective findings inconclusive? and (3) where should neuroimaging research go from here? It is noted that the vast majority of MRI scans in autism are clinically…
Descriptors: Autism, Clinical Diagnosis, Etiology, Neurology
King, Bryan H.; And Others – American Journal on Mental Retardation, 1991
This commentary critiques two papers by Curt Sandman, pointing out interpretive problems in models explaining self-injurious behavior in terms of opioids. Withdrawal effects are emphasized as an alternative to hypotheses asserting congenital opioid excess as a cause of sensory depression or an addiction to a relative excess of opioid activity in…
Descriptors: Behavior Disorders, Biochemistry, Etiology, Models
Demet, Edward M.; Sandman, Curt A. – American Journal on Mental Retardation, 1991
This paper answers criticism (EC 601 030) of the authors' work regarding opioid explanations of self-injurious behavior. Possible withdrawal effects are ruled out as an explanation, in favor of opioid excess leading to sensory depression and addiction to relative excesses of opioid activity in the brain. Alternative models of consequences of…
Descriptors: Behavior Disorders, Biochemistry, Etiology, Models

Wong, Virginia; Wong, Sik Nin – Journal of Autism and Developmental Disorders, 1991
Brainstem auditory evoked potentials were compared in 109 children with infantile autism, 38 with autistic condition, 19 with mental retardation, and 20 normal children. Children with infantile autism or autistic condition had significantly longer brainstem transmission time than normal children suggesting neurological damage as the basis of…
Descriptors: Autism, Children, Etiology, Neurological Impairments

Mulaik, Stanley A. – Child Development, 1987
Examines and rejects common criticisms of the causality concept; shows causality is a relation implied in the grammar of a language about objects. Discusses objective criteria for concepts of causal relations and explains how the concept of causality may be modified to have causes determine probabilities of outcomes. (Author/RH)
Descriptors: Definitions, Etiology, Probability, Research Methodology

LaBuda, Michele C.; DeFries, J. C. – Annals of Dyslexia, 1988
Data analysis of 134 twin pairs from the Colorado Reading Project found that approximately 40 percent of the deficit observed in disabled readers is because of genetic factors, 35 percent because of environmental influences shared by twin pairs, and 25 percent because of environmental factors unique to the individual and/or error variance.…
Descriptors: Environmental Influences, Etiology, Genetics, Predictive Measurement

Hall, Joseph W.; Grose, John H. – Journal of Speech and Hearing Research, 1994
This study investigated comodulation masking release (CMR) in 42 children (ages 5 to 10) with a history of otitis media with effusion (OME) and 19 control children with no history of ear disease. Results indicated that monotic CMRs were reduced in children with hearing loss due to OME and these CMRs remained abnormally small for several months.…
Descriptors: Auditory Evaluation, Children, Ears, Etiology

Perkins, William H.; And Others – Journal of Speech and Hearing Research, 1991
A theory of neurolinguistic function is proposed to explain fluency and the production of stuttered speech disruptions. Stuttering results when the speaker is under time pressure and is unaware of the cause of dyssynchrony between the linguistic and paralinguistic components of speech which are processed by different neural systems but converge on…
Descriptors: Etiology, Expressive Language, Linguistics, Neurology

Ingham, Roger J.; And Others – Journal of Speech and Hearing Research, 1996
Analysis of use of positron emission tomographic measurements of resting-state regional cerebral blood flow in 29 men, 10 of whom stuttered, did not support the idea that developmental stuttering is associated with abnormalities of blood flow at rest. Findings did suggest an essentially normal functional brain terrain with a small number of minor…
Descriptors: Adults, Brain Hemisphere Functions, Etiology, Males